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![PDF] Congenital adrenal hyperplasia due to 21-hydroxylase deficiency – management and differentiation in adults | Semantic Scholar](https://d3i71xaburhd42.cloudfront.net/60d7869a604000dafce2a0ce0f8f8a59e20ef65e/3-Figure1-1.png "PDF] Congenital adrenal hyperplasia due to 21-hydroxylase deficiency – management and differentiation in adults | Semantic Scholar")
PDF] Congenital adrenal hyperplasia due to 21-hydroxylase deficiency – management and differentiation in adults | Semantic Scholar
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![Figure 1 from [Congenital adrenal hyperplasia due to 21-hydroxylase deficiency--management in adults]. | Semantic Scholar](https://d3i71xaburhd42.cloudfront.net/168c37ff0c4cd5b38b0d0ceb6d096bdaa45415da/3-Figure1-1.png "Figure 1 from [Congenital adrenal hyperplasia due to 21-hydroxylase deficiency--management in adults]. | Semantic Scholar")
Figure 1 from [Congenital adrenal hyperplasia due to 21-hydroxylase deficiency--management in adults]. | Semantic Scholar
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Osmosis from Elsevier - Today we're tackling a tough one - congenital adrenal enzyme deficiencies, which are 17 alpha, 21 alpha, and 11 beta hydroxylase deficiency. All three affect cortisol production and
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